De ce oamenii nu pot trăi până la 200 de ani, deși ficatul ar rezista milenii?

A recent study suggests humans could theoretically live up to 156 years if aging mechanisms were reversed, but somatic mutations limit lifespan. These mutations accumulate in non-regenerative cells like neurons and heart muscle, affecting tissue function. The research indicates that while advanced anti-aging therapies exist, they cannot address the genetic changes that occur over a lifetime. The study highlights the importance of understanding somatic mutations in aging and suggests future therapies should focus on more accessible targets.